Non-structural 5A resistance-associated substitutions and Interleukin28B in HCV genotype 3b decompensated cirrhosis patients with sofosbuvir plus velpatasvir
1.1. Background: Hepatitis C Virus (HCV) Genotype (GT) infection is still regarded as one of the more “difficult-to-treat” GTs in the era of IFN-free treatments. The viral factors involved in this unfavorable outcome have yet to be clarified.